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At a distance of 40-45 miles, a person would have at most 3 hours after the fallout began to find shelter. Considerably smaller radiation doses will make people seriously ill. Thus, the survival prospects of persons immediately downwind of the burst point would be slim unless they could be sheltered or evacuated.
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Read More »Both the local and worldwide fallout hazards of nuclear explosions depend on a variety of interacting factors: weapon design, explosive force, altitude and latitude of detonation, time of year, and local weather conditions. All present nuclear weapon designs require the splitting of heavy elements like uranium and plutonium. The energy released in this fission process is many millions of times greater, pound for pound, than the most energetic chemical reactions. The smaller nuclear weapon, in the low-kiloton range, may rely solely on the energy released by the fission process, as did the first bombs which devastated Hiroshima and Nagasaki in 1945. The larger yield nuclear weapons derive a substantial part of their explosive force from the fusion of heavy forms of hydrogen--deuterium and tritium. Since there is virtually no limitation on the volume of fusion materials in a weapon, and the materials are less costly than fissionable materials, the fusion, "thermonuclear," or "hydrogen" bomb brought a radical increase in the explosive power of weapons. However, the fission process is still necessary to achieve the high temperatures and pressures needed to trigger the hydrogen fusion reactions. Thus, all nuclear detonations produce radioactive fragments of heavy elements fission, with the larger bursts producing an additional radiation component from the fusion process. The nuclear fragments of heavy-element fission which are of greatest concern are those radioactive atoms (also called radionuclides) which decay by emitting energetic electrons or gamma particles. (See "Radioactivity" note.) An important characteristic here is the rate of decay. This is measured in terms of "half-life"--the time required for one-half of the original substance to decay--which ranges from days to thousands of years for the bomb-produced radionuclides of principal interest. (See "Nuclear Half-Life" note.) Another factor which is critical in determining the hazard of radionuclides is the chemistry of the atoms. This determines whether they will be taken up by the body through respiration or the food cycle and incorporated into tissue. If this occurs, the risk of biological damage from the destructive ionizing radiation (see "Radioactivity" note) is multiplied. Probably the most serious threat is cesium-137, a gamma emitter with a half-life of 30 years. It is a major source of radiation in nuclear fallout, and since it parallels potassium chemistry, it is readily taken into the blood of animals and men and may be incorporated into tissue. Other hazards are strontium-90, an electron emitter with a half-life of 28 years, and iodine-131 with a half-life of only 8 days. Strontium-90 follows calcium chemistry, so that it is readily incorporated into the bones and teeth, particularly of young children who have received milk from cows consuming contaminated forage. Iodine-131 is a similar threat to infants and children because of its concentration in the thyroid gland. In addition, there is plutonium-239, frequently used in nuclear explosives. A bone-seeker like strontium-90, it may also become lodged in the lungs, where its intense local radiation can cause cancer or other damage. Plutonium-239 decays through emission of an alpha particle (helium nucleus) and has a half-life of 24,000 years. To the extent that hydrogen fusion contributes to the explosive force of a weapon, two other radionuclides will be released: tritium (hydrogen-3), an electron emitter with a half-life of 12 years, and carbon-14, an electron emitter with a half-life of 5,730 years. Both are taken up through the food cycle and readily incorporated in organic matter. Three types of radiation damage may occur: bodily damage (mainly leukemia and cancers of the thyroid, lung, breast, bone, and gastrointestinal tract); genetic damage (birth defects and constitutional and degenerative diseases due to gonodal damage suffered by parents); and development and growth damage (primarily growth and mental retardation of unborn infants and young children). Since heavy radiation doses of about 20 roentgen or more (see "Radioactivity" note) are necessary to produce developmental defects, these effects would probably be confined to areas of heavy local fallout in the nuclear combatant nations and would not become a global problem.
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Read More »This means that between 3 and 50 persons per billion births in the post-testing generation will have genetic damage for each megaton of nuclear yield exploded. With similar uncertainty, it is possible to estimate that the induction of cancers would range from 75 to 300 cases per megaton for each billion people in the post-test generation. If we apply these very rough yardsticks to a large-scale nuclear war in which 10,000 megatons of nuclear force are detonated, the effects on a world population of 5 billion appear enormous. Allowing for uncertainties about the dynamics of a possible nuclear war, radiation-induced cancers and genetic damage together over 30 years are estimated to range from 1.5 to 30 million for the world population as a whole. This would mean one additional case for every 100 to 3,000 people or about 1/2 percent to 15 percent of the estimated peacetime cancer death rate in developed countries. As will be seen, moreover, there could be other, less well understood effects which would drastically increase suffering and death.
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